Max in WT synaptoneurosomes, suggesting that Src signaling could be downregulated in KI synapses. Alternatively, our capability to rescue SERT purpose in KI midbrain synaptoneurosomes through the inhibition of FAK suggests elevated FAK signaling downstream with the Pro32Pro33 mutant, as verified by greater pFAK localization in five-HT synapses. Our data https://damienznykt.develop-blog.com/39021689/pro33-secrets